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Mide ve duodenum · Patoloji · Orta öncelik

Portal hipertansif gastropati ve gastrik varis görünümü

Portal hypertensive gastropathy and gastric varices

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Portal hipertansif gastropati ve gastrik varis görünümü: yayımlanmış olgu görüntüsü, aksiyel kesit
Görüntü konuyu kısmen gösteriyor; bulgunun bir bölümü seçilebilir.

4 adım

Görüntü: Graham DW, Thompson B, Mantry P., “Unusual Presentation of Von Hippel-Lindau Syndrome With Gastric Variceal Bleeding.” 2024, Figure 1. PMC11327004 · doi:10.7759/cureus.64685 · CC BY 4.0. Değişiklik: yeniden boyutlandırıldı.

Özet

Siroz veya portal venöz akım bozukluğu olan hastada mide mukozasındaki konjesyon ve gastrik varisler BT'de portal hipertansiyonun eşlik eden bulguları olarak görülebilir. Portal faz kontrastlı BT, mide duvarı ve kıvrımların yanı sıra submukozal/perigastrik kollateralleri, besleyici venleri ve gastrorenal şantı haritalar. BT mukozal gastropati tanısını tek başına koydurmaz; portal hipertansif gastropatinin esas tanısı endoskopik görünümle konur. Varislerin kıvrımlı damarlar yerine kitle veya kalın mide kıvrımı sanılması kanama riskinin ve işlem öncesi damar anatomisinin yanlış değerlendirilmesine neden olabilir.

Faz ve pencere

Arteriyel
Dinamik karaciğer incelemesinde fundus veya korpus mukozasında geçici segmental/subsegmental hipoattenüasyon görülüp portal veya denge fazında normale dönebilir; bu işaret PHG ile ilişkilendirilmiştir, ancak PHG tanısı endoskopik görünümle konur ve BT bulgusu endoskopinin yerini tutmaz.
Portal tanısal
Portal ven ve mezenterik venlerle benzer yoğunlukta kontrastlanan, mide duvarı/submukozası içinde veya perigastrik alanda tübüler-serpijinöz venöz kanallar; splenik/sol gastrik ven bağlantısı, gastrorenal şant, splenomegali, asit ve karaciğer morfolojisi değerlendirilir.

Önerilen pencereler: Batın (G 400 / M 50), Karaciğer (G 150 / M 30), Anjiyo (G 600 / M 150).

BT bulguları

  • Fundus ve korpus kıvrımlarının kalın görünümü tek başına PHG tanısı koydurmaz; PHG, bu bölgelerdeki karakteristik endoskopik bulgularla tanınır.
  • Mide duvarı/submukozası içinde portal venle aynı düzeyde kontrastlanan kıvrımlı venöz kanallar
  • Mide fundusu çevresinde kısa gastrik ve posterior gastrik venlerden uzanan serpijinöz perigastrik kollateraller
  • Küçük kurvatur boyunca sol gastrik (koroner) ven ve gastroözofageal bileşkede kollateral damarlar
  • Fundal kollaterallerin sol renal vene açıldığı gastrorenal şant
  • Splenomegali, asit, portal ven kalibrasyon/değişiklikleri ve diğer portosistemik kollateraller

Ölçütler ve sınıflamalar

Sarin gastrik varis sınıflaması (endoskopik topografi)
GOV1: özofagus varisinin küçük kurvatur boyunca uzantısı; GOV2: özofagogastrik bileşkedeki varisin fundus yönünde uzanımı. IGV1: fundusta izole varis; IGV2: fundus dışındaki mide bölgelerinde izole varis. Sınıflama endoskopik yerleşim tanımıdır; BT'de damarların topografisini tarif etmeye yardımcı olur.

Normalde

Normal midede fundus ve korpus kıvrımları düzenlidir; duvar içinde portal venle eş kontrastlanan serpijinöz submukozal damar ağı veya perigastrik kollateral kümesi görülmez. Kıyaslamada mukoza/kıvrım görünümünü, kısa gastrik ve sol gastrik venlerin seyrini, portal venöz fazda damarlarla eş zamanlı kontrastlanan kıvrımlı kanalların duvar içinde mi dışında mı olduğunu belirle.

Normal BT ile kıyasla

Aynı bölgenin, kaynak veri setinde patoloji içermediği belirtilen bir BT incelemesini kesit kesit kaydırın; organ sınırlarını açarak anatomiyi hasta görüntüsüyle karşılaştırın. Küçük rastlantısal bulgular tümüyle dışlanmamıştır.

Kaynak: TotalSegmentator v2.01 veri seti (Wasserthal ve ark., Radiology: Artificial Intelligence 2023), CC BY 4.0, vaka s0541; organ sınırları veri setinin otomatik segmentasyonundan, birleştirilerek sadeleştirildi. Görüntüler 2,5 mm kesit aralığına yeniden örneklendi.

Ayırıcı tanı

Gastrik adenokarsinom
fokal düzensiz duvar kitlesi ve lümen deformasyonu; portal kanla eş yoğunlukta damar kanalları oluşturmaz
GIST
ekzofitik veya intramural solid kitle; damar gibi tübüler ve dallanan seyir göstermez
Gastrik rugae hipertrofisi
duvar kıvrımıyla devamlı yumuşak doku; portal fazda ayrı ayrı kontrastlanan lümenli kanallar değildir
Gastrit ve PHG
mukozal ödem ve diffüz hiperkontrastlanmaya yol açabilir; ancak varislerdeki gibi lümenli, portal ven yoğunluğunda düğümlenmiş veya serpijinöz kanal yapıları oluşturmaz.
Sol taraflı portal hipertansiyon, splenik ven trombozu gibi splanchnik venöz tıkanıklıklarla ilişkili olabilir; gastrik varisler portal hipertansiyonda görülebilir.

Tuzaklar

  • Gastrik varisler endoskopi veya baryum incelemesinde tümör ya da kalınlaşmış mide kıvrımlarını taklit edebilir; kontrastlı BT'de submukozal ve perigastrik kollateraller damar yapıları olarak görülebilir.
  • Gastrik varis ile perigastrik kollateral aynı yerde bulunabilir ama biri duvar içi, diğeri dış yüzeydedir; raporda anatomik kompartımanı tarif et.
  • Portal hipertansif gastropatinin mukozal görünümü endoskopik tanıdır; BT'de duvar ödemi veya arteriyel perfüzyon kusuru tek başına kesin PHG sayılmaz.
  • Fundal varisler siroz olmaksızın splenik ven darlığı veya tıkanıklığıyla ilişkili sol taraflı portal hipertansiyonda görülebilir; BT'de splenik ven ve portal sistem bulgularını karaciğer morfolojisiyle birlikte değerlendir.

Kendini dene

  1. Sirozlu hastada fundus duvarında portal venle eş kontrastlanan serpijinöz yapılar ve sol renal vene devamlılık izleniyor. En olası nedir?

    Cevabı göster

    Gastrik varis ve gastrorenal şant. Portal venle eş kontrastlanan kıvrımlı submukozal damarlar ve renal vene devamlılık gastrik varis/gastrorenal şantı gösterir. Gastrik varisler BT'de tümörü veya kalınlaşmış rugae'yi taklit edebilir; portal ve mezenterik venlerle benzer kontrastlanan tübüler ya da serpijinöz damarlar varis lehine bulgudur.

  2. GOV2 ve IGV1 gastrik varisleri genellikle hangi venlerden beslenir?

    Cevabı göster

    Kısa ve posterior gastrik venler. GOV2 ve IGV1 genellikle kısa ve posterior gastrik venlerden beslenir. GOV1 ise sol gastrik venin ön dalıyla ilişkilidir.

Kaynaklar

Bu sayfadaki 39 cümle ve 2 quiz sorusu aşağıdaki kaynaklardan alıntılarla otomatik olarak karşılaştırıldı; 8 cümle bu karşılaştırmada düzeltildi (2026-10-09).

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Eğitim amaçlıdır; hasta başında tanı ve tedavi kararının yerine geçmez, tıbbi tavsiye değildir. Metinler yapay zekâ desteğiyle yazıldı ve otomatik bir adımda kaynak alıntılarıyla karşılaştırıldı; bu bir tıbbi doğrulama değildir ve içerik uzman radyolog incelemesinden geçmedi. Klinik kararlarda güncel kılavuzları, kurum protokollerini ve radyoloji raporunu esas alın.